Fish Scale Cocaine

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What is Cocaine: 

Fish scale cocaine (derived from the French word “cocaïne,” which traces back to the Spanish “coca” and ultimately from the Quechua term “kúka”) is a powerful stimulant of the central nervous system (CNS), extracted from the leaves of two South American plants, Erythroxylum coca and Erythroxylum novogranatense.

Once the cocaine is extracted from coca leaves and processed into cocaine hydrochloride (commonly known as powdered cocaine), it can be consumed in various ways: snorted, heated until it sublimates and then inhaled, or dissolved and injected into a vein. Cocaine primarily acts by stimulating the brain’s reward system.

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It has been suggested as an aid in the treatment of cocaine dependence. In one controversial study, an infusion of coca leaves, combined with counseling, was used to treat 23 individuals addicted to smoking coca paste in Lima, Peru.

The number of relapses dropped from an average of four times per month before treatment with coca tea to just one during the treatment period. Fish scale cocaine.

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When the Spanish first arrived in South America, they largely dismissed indigenous claims that chewing coca leaves provided strength and energy, attributing the practice to the work of the Devil. However, after realizing the truth of these claims, they legalized and taxed the leaf, taking a 10% cut from every crop.

“When the chemical leaves the body and the brain tries to restore its chemical balance, this leads to the ‘comedown,’ during which the user feels drained, depressed or irritable, anxious, or paranoid,” explains Nutt. The more chemicals are cleared, the stronger the urge to replace them. “Some users find it difficult to resist taking more cocaine to delay the inevitable comedown,” Nutt adds.

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Coca leaves have been found alongside ancient Peruvian mummies, and pottery from that era shows people with bulging cheeks, suggesting they were chewing something, likely coca leaves. Evidence also indicates that these cultures used a combination of coca leaves and saliva as an anesthetic during trepanation procedures.

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With the discovery of this new alkaloid, Western medicine quickly explored the potential uses of the plant.

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Although the stimulant and appetite-suppressant effects of coca leaves were known for centuries, the isolation of the cocaine alkaloid wasn’t achieved until 1855. Several European scientists attempted to extract cocaine, but their efforts were unsuccessful due to two main factors:

1. The level of chemical knowledge available at the time was insufficient, and
2. The conditions of sea transport from South America often led to the degradation of the cocaine in the plant samples sent to European chemists. Buy pure Columbia cocaine online.

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Side Effects of Cocaine Abuse

Mental effects of cocaine may involve an intense sense of euphoria, heightened sexual arousal, disconnection from reality, or agitation. Physically, it can lead to a rapid heartbeat, sweating, and dilated pupils. High doses might cause elevated blood pressure or increased body temperature. The effects typically start within seconds to minutes of use and can last from five to ninety minutes.

Cocaine’s numbing and vasoconstrictive properties make it useful in surgical procedures involving the throat or nasal passages to manage pain, reduce bleeding, and prevent vocal cord spasms. The drug crosses the blood-brain barrier primarily through a proton-coupled organic cation antiporter and, to a lesser extent, by passive diffusion through cell membranes.

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Cocaine interferes with the dopamine transporter, preventing the reuptake of dopamine from the synaptic cleft into the pre-synaptic axon terminal. This leads to elevated dopamine levels in the synaptic cleft, which enhances activation of dopamine receptors in the post-synaptic neuron, resulting in feelings of euphoria and heightened arousal.

Additionally, cocaine blocks the serotonin and norepinephrine transporters, stopping the reuptake of serotonin and norepinephrine from the synaptic cleft into the pre-synaptic axon terminal. This increases the activation of serotonin and norepinephrine receptors in the post-synaptic neuron, contributing to the overall mental and physical effects experienced with cocaine use.

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A single dose of cocaine can lead to immediate tolerance to its effects, and repeated use may result in addiction. Individuals who stop using cocaine often experience cravings and withdrawal symptoms, which include depression, reduced libido, diminished ability to experience pleasure, and fatigue.

Cocaine use heightens the risk of death overall, with intravenous use especially increasing the chances of trauma and infections such as bloodborne diseases and HIV.

It also raises the risk of stroke, heart attack, irregular heartbeats, lung damage (particularly when smoked), and sudden cardiac death. Illicit cocaine is often mixed with substances like local anesthetics, levamisole, cornstarch, quinine, or sugar, which can introduce additional toxicity.

According to the Global Burden of Disease study in 2017, cocaine use was responsible for approximately 7,300 deaths annually worldwide.

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